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LV dilatation in ICM usually occurs due to adverse remodelling after transmural myocardial infarction. During echocardiography, this is manifested by thinned, akinetic myocardial segments in the infarcted territory with cavity dilatation and mild compensatory hypertrophy of the non-infarcted segments.

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Cardiac MRI showed a moderately dilated LV with mild-to-moderate concentric LV hypertrophy with wall thickness of 13 mm. LV systolic function was severely reduced with quantitative EF of 22% and severe global hypokinesis.

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